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Thank you for the explanation. I believe the problem is the fact that your server is being hit by a Denial of Service Attack, if so you need to look at the logs on your server. There may be some specific logs in there that your server is generating that your script is not.
For example, I use Webmin, and it’s able to tell me if a DOS is coming from a specific IP address, however the only logs available for me to review are for every time the Webserver is run, and I wouldn’t know how to find out where the attack is coming from without someone with some specific knowledge of your situation doing it.
If you’re on a shared hosting account, you can’t get access to your server’s logs, so the only thing I can think of is checking if the script is working in a local environment on your server.
This is an application for a LUND «Cancer in Pets/CIP» grant to examine the role of the fat stored in the adipose tissue in the development of lung cancer in dogs. Canine adrenal cortical carcinoma (ACC), the most common form of malignant lung cancer in dogs, has been the focus of previous grants in which the hypothesis has been explored that the cells that comprise the primary and metastatic tumors are clonal reproductions of a single common stem cell. This hypothesis proposes that the occurrence of tumors in many different tissues and their differentiation into multiple cells expressing different phenotypes are due to a single progenitor cell that is present in the tissue in which the tumor develops, and that those cells in tumors differentiating into cell phenotypes different from the original progenitor become lost during tumor progression. To test this hypothesis, we have established a colony-forming cell assay using liposuctioned adipose tissue and seeding this in a 3-dimensional collagen matrix. The cells from these colonies, generated in a similar environment as that for the cancer, can differentiate into the different lineages of cells including fibroblasts, adipocytes, chondrocytes, osteocytes, smooth muscle cells, granulocytes, and macrophages. We have detected the presence of cells from the same clones in both primary and metastatic tumors, and in the metastatic tumors the cells that have differentiated into cell phenotypes different from the original progenitor have been lost. This indicates that metastasis is not the result of a branching or divergent pathway in the tumor clone, but
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